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The main signaling cascade for local translation at spines requires glutamate binding to metabotropic glutamate receptors (mGluR), which triggers protein-synthesis-dependent forms of spine plasticity by activating extracellular signal-regulated kinase (ERK) or mammalian target of rapamycin (mTOR) pathways (Ebert and Greenberg, Camk2a , Actb , or Limk1

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Importantly, liver-specific insulin receptor-deficient mice (LIRKO mice) exhibit hyperglycemia associated with increased G6pc expression 7